y drivers of CPUO, underscoring the role of IL-31
Galderma’s study builds on a recent investigation into the causes of inflammation in CPUO, which uncovered critical insights into its complex inflammatory profile. The research – presented at the Society of Investigative Dermatology annual meeting in San Diego in May 2025 – found a significant increase in IL-31-producing CD4+ T cells in CPUO patients, reinforcing IL-31 as a key driver of the disease.9These results open the door to targeted therapies that address the root causes of CPUO, a disease with significant unmet needs that currently has no approved treatment options.1,9








“The first patient enrollment in this study marks an important milestone in our commitment to advancing dermatology for every skin story – especially in areas of high unmet need. CPUO is a deeply distressing condition for patients, and the absence of appr
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